Postinflammatory hyperpigmentation
Postinflammatory hyperpigmentation は、皮膚の炎症後に色素が増加することを特徴とする状態です。Postinflammatory hyperpigmentation は、長時間の日光曝露、炎症、座瘡に関連するものなど、その他の皮膚損傷によって引き起こされる可能性があります。肌の色が濃い人は、過度に日光にさらされると色素沈着が過剰になる傾向があります。
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ドイツの2022年Stiftung Warentestの結果では、ModelDermに対する消費者満足度は有料の遠隔医療相談よりもわずかに低いだけでした。
ドイツの2022年Stiftung Warentestの結果では、ModelDermに対する消費者満足度は有料の遠隔医療相談よりもわずかに低いだけでした。
relevance score : -100.0%
ReferencesPostinflammatory hyperpigmentation (PIH) は、皮膚の炎症や外傷の後に起こる一般的な皮膚トラブルです。この症状は長期間続くことが多く、肌の色が濃い人ではさらに悪化しやすいです (Fitzpatrick skin types III–VI)。自然に改善することもありますが、回復までに時間がかかるため、長期的な治療が必要になることも少なくありません。さまざまな治療法を組み合わせることが最も効果的です。
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation は、皮膚炎症の一般的な続発症です。肌の色が濃いほど、重篤で頻繁に影響を受けやすい傾向があります。研究によると、Postinflammatory hyperpigmentation のような問題は、肌の色が濃い人が皮膚科の受診を求める主な理由の一つです。Postinflammatory hyperpigmentation を解決するには早期の治療が重要で、通常は初期の炎症状態を管理することから始めます。最初の治療では、皮膚を明るくする局所剤と、保護のための日焼け止めを併用します。hydroquinone、azelaic acid、kojic acid、arbutin、licorice extracts などの薬剤は、過剰な色素沈着を効果的に軽減します。さらに、retinoids、mequinol、ascorbic acid、niacinamide、N‑acetyl glucosamine、soy も色素脱失剤として使用されており、新しい治療法が続々と登場しています。通常、表面的な色素沈着過剰には局所治療が効果的ですが、頑固な場合にはレーザーや化学ピーリングなどの処置が必要になることがあります。Postinflammatory hyperpigmentation の炎症や悪化を防ぐため、これらの治療を行う際は注意が必要です。
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
